Analysis of autophagy in DLBCL reveals subtype-specific differences and the preferential targeting of ULK1 inhibition in GCB-DLBCL provides a rationale as a new therapeutic approach.

Mandhair, Harpreet KaurHarpreet KaurMandhairRadpour, RaminRaminRadpourWesterhuis, MiraMiraWesterhuisBanz Wälti, Yara SarahYara SarahBanz Wälti0000-0003-4474-3132Humbert, MagaliMagaliHumbert0000-0003-3820-0297Arambasic, MiroslavMiroslavArambasicDengjel, JörnJörnDengjelDavies, AndrewAndrewDaviesTschan, Mario PaulMario PaulTschan0000-0001-5897-3647Novak, UrbanUrbanNovak2024-10-262024-10-262024-02https://boris-portal.unibe.ch/handle/20.500.12422/173737en600 - Technology::610 - Medicine & health500 - Science::570 - Life sciences; biologyAnalysis of autophagy in DLBCL reveals subtype-specific differences and the preferential targeting of ULK1 inhibition in GCB-DLBCL provides a rationale as a new therapeutic approach.article10.48350/1920573826343110.1038/s41375-024-02147-4