Publication:
Arginase II Promotes Macrophage Inflammatory Responses Through Mitochondrial Reactive Oxygen Species, Contributing to Insulin Resistance and Atherogenesis

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cris.virtualsource.author-orcid3052ae31-b9ca-4adc-b818-168d118adc57
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dc.contributor.authorMing, Xiu-Fen
dc.contributor.authorRajapakse, Angana G
dc.contributor.authorYepuri, Gautham
dc.contributor.authorXiong, Yuyan
dc.contributor.authorCarvas, João M
dc.contributor.authorRuffieux, Jean
dc.contributor.authorScerri, Isabelle
dc.contributor.authorWu, Zongsong
dc.contributor.authorPopp, Katja
dc.contributor.authorLi, Jianhui
dc.contributor.authorSartori, Claudio
dc.contributor.authorScherrer, Urs
dc.contributor.authorKwak, Brenda R
dc.contributor.authorMontani, Jean-Pierre
dc.contributor.authorYang, Zhihong
dc.date.accessioned2024-10-13T12:58:26Z
dc.date.available2024-10-13T12:58:26Z
dc.date.issued2012
dc.description.abstractMacrophage-mediated chronic inflammation is mechanistically linked to insulin resistance and atherosclerosis. Although arginase I is considered antiinflammatory, the role of arginase II (Arg-II) in macrophage function remains elusive. This study characterizes the role of Arg-II in macrophage inflammatory responses and its impact on obesity-linked type II diabetes mellitus and atherosclerosis.
dc.description.numberOfPages1
dc.description.sponsorshipUniversitätsklinik für Kardiologie
dc.identifier.pmid23130157
dc.identifier.urihttps://boris-portal.unibe.ch/handle/20.500.12422/89356
dc.language.isoen
dc.publisherAmerican Heart Association
dc.publisher.placeNew York, N.Y.
dc.relation.ispartofJournal of the American Heart Association
dc.relation.issn2047-9980
dc.relation.organizationDCD5A442BB15E17DE0405C82790C4DE2
dc.titleArginase II Promotes Macrophage Inflammatory Responses Through Mitochondrial Reactive Oxygen Species, Contributing to Insulin Resistance and Atherogenesis
dc.typearticle
dspace.entity.typePublication
oaire.citation.issue4
oaire.citation.startPagee000992
oaire.citation.volume1
oairecerif.author.affiliationUniversitätsklinik für Kardiologie
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unibe.description.ispublishedpub
unibe.eprints.legacyId15297
unibe.journal.abbrevTitleJ Am Heart Assoc
unibe.subtype.articlejournal

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