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  3. The Gut-Brain Axis: How Microbiota and Host Inflammasome Influence Brain Physiology and Pathology.
 

The Gut-Brain Axis: How Microbiota and Host Inflammasome Influence Brain Physiology and Pathology.

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BORIS DOI
10.48350/160574
Date of Publication
December 2020
Publication Type
Article
Division/Institute

Universitätsklinik fü...

Department for BioMed...

Contributor
Rutsch, Andrina
Universitätsklinik für Viszerale Chirurgie und Medizin, Gastroenterologie
Department for BioMedical Research, Forschungsgruppe Gastroenterologie / Mukosale Immunologie
Kantsjö, Björn Johan
Department for BioMedical Research, Forschungsgruppe Gastroenterologie / Mukosale Immunologie
Universitätsklinik für Viszerale Chirurgie und Medizin, Gastroenterologie
Ronchi, Francesca
Department for BioMedical Research, Forschungsgruppe Gastroenterologie / Mukosale Immunologie
Universitätsklinik für Viszerale Chirurgie und Medizin, Gastroenterologie
Subject(s)

600 - Technology::610...

Series
Frontiers in immunology
ISSN or ISBN (if monograph)
1664-3224
Publisher
Frontiers Research Foundation
Language
English
Publisher DOI
10.3389/fimmu.2020.604179
PubMed ID
33362788
Uncontrolled Keywords

Alzheimer’s disease P...

Description
The human microbiota has a fundamental role in host physiology and pathology. Gut microbial alteration, also known as dysbiosis, is a condition associated not only with gastrointestinal disorders but also with diseases affecting other distal organs. Recently it became evident that the intestinal bacteria can affect the central nervous system (CNS) physiology and inflammation. The nervous system and the gastrointestinal tract are communicating through a bidirectional network of signaling pathways called the gut-brain axis, which consists of multiple connections, including the vagus nerve, the immune system, and bacterial metabolites and products. During dysbiosis, these pathways are dysregulated and associated with altered permeability of the blood-brain barrier (BBB) and neuroinflammation. However, numerous mechanisms behind the impact of the gut microbiota in neuro-development and -pathogenesis remain poorly understood. There are several immune pathways involved in CNS homeostasis and inflammation. Among those, the inflammasome pathway has been linked to neuroinflammatory conditions such as multiple sclerosis, Alzheimer's and Parkinson's diseases, but also anxiety and depressive-like disorders. The inflammasome complex assembles upon cell activation due to exposure to microbes, danger signals, or stress and lead to the production of pro-inflammatory cytokines (interleukin-1β and interleukin-18) and to pyroptosis. Evidences suggest that there is a reciprocal influence of microbiota and inflammasome activation in the brain. However, how this influence is precisely working is yet to be discovered. Herein, we discuss the status of the knowledge and the open questions in the field focusing on the function of intestinal microbial metabolites or products on CNS cells during healthy and inflammatory conditions, such as multiple sclerosis, Alzheimer's and Parkinson's diseases, and also neuropsychiatric disorders. In particular, we focus on the innate inflammasome pathway as immune mechanism that can be involved in several of these conditions, upon exposure to certain microbes.
Handle
https://boris-portal.unibe.ch/handle/20.500.12422/54014
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fimmu-11-604179.pdftextAdobe PDF1.52 MBpublishedOpen
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