Publication:
Disruption of Notch1 induces vascular remodeling, intussusceptive angiogenesis, and angiosarcomas in livers of mice

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cris.virtual.author-orcid0000-0002-5062-1169
cris.virtualsource.author-orcid50f55964-7ff8-4bc0-8549-9919a3cbee93
cris.virtualsource.author-orcid6b9f7e28-8a66-49ee-abac-5a92d89b810b
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dc.contributor.authorDill, Michael T
dc.contributor.authorRothweiler, Sonja
dc.contributor.authorDjonov, Valentin Georgiev
dc.contributor.authorHlushchuk, Ruslan
dc.contributor.authorTornillo, Luigi
dc.contributor.authorTerracciano, Luigi
dc.contributor.authorMeili-Butz, Silvia
dc.contributor.authorRadtke, Freddy
dc.contributor.authorHeim, Markus H
dc.contributor.authorSemela, David
dc.date.accessioned2024-10-11T09:30:51Z
dc.date.available2024-10-11T09:30:51Z
dc.date.issued2012
dc.description.abstractNotch signaling mediates embryonic vascular development and normal vascular remodeling; Notch1 knockout mice develop nodular regenerative hyperplasia (NRH). The pathogenesis of NRH is unclear, but has been associated with vascular injury in the liver sinusoids in clinical studies. We investigated the role of Notch1 signaling in liver sinusoidal endothelial cells (LSECs).
dc.description.numberOfPages12
dc.description.sponsorshipInstitut für Anatomie
dc.identifier.isi000302296400046
dc.identifier.pmid22245843
dc.identifier.publisherDOI10.1053/j.gastro.2011.12.052
dc.identifier.urihttps://boris-portal.unibe.ch/handle/20.500.12422/78575
dc.language.isoen
dc.publisherElsevier
dc.publisher.placePhiladelphia, Pa.
dc.relation.ispartofGastroenterology
dc.relation.issn0016-5085
dc.relation.organizationDCD5A442BCD7E17DE0405C82790C4DE2
dc.subject.ddc600 - Technology::610 - Medicine & health
dc.titleDisruption of Notch1 induces vascular remodeling, intussusceptive angiogenesis, and angiosarcomas in livers of mice
dc.typearticle
dspace.entity.typePublication
oaire.citation.endPage977.e2
oaire.citation.issue4
oaire.citation.startPage967
oaire.citation.volume142
oairecerif.author.affiliationInstitut für Anatomie
oairecerif.author.affiliationInstitut für Anatomie
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unibe.description.ispublishedpub
unibe.eprints.legacyId8132
unibe.journal.abbrevTitleGASTROENTEROLOGY
unibe.refereedtrue
unibe.subtype.articlejournal

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