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  3. Exercise-induced modulation of kynurenine pathway metabolite patterns in multiple sclerosis: a targeted metabolomics analysis across four randomized controlled trials.

Exercise-induced modulation of kynurenine pathway metabolite patterns in multiple sclerosis: a targeted metabolomics analysis across four randomized controlled trials.

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DOI
10.48620/99905
Publisher DOI
10.1016/j.bbi.2026.106930
PubMed ID
42526662
Abstract
Kynurenine pathway imbalance is a characteristic feature of multiple sclerosis (MS). Serum kynurenines contribute to latent factors reflecting a neurotoxic (NeuroTox) or neuroprotective pattern (NeuroPro). Previous single-metabolite studies show a favorable modulation of the kynurenine pathway by acute exercise, characterized by increased neuroprotective kynurenic acid. This study aims to investigate the acute and chronic effects of different exercise modalities on NeuroTox and NeuroPro. In this pooled analysis of randomized controlled trials, we followed four subcohorts of our previous work, in which we identified NeuroTox and NeuroPro. Serum concentrations of kynurenines were quantified by liquid chromatography-tandem mass spectrometry. Pattern stability was assessed by repeating exploratory factor analysis (EFA). Acute effects of a single session of combined strength-focused exercise (CE; FunTraiMS study) and chronic effects of exercise interventions, including CE (10 weeks; FunTraiMS study) and endurance exercise (3 weeks; HIITMS study and 24 weeks; EMSES and EXBRAIN studies), were evaluated using linear mixed-effects models. The four subcohorts included 273 participants (67.0% female; mean (SD) age: 44.5 (10.6) years). Repeated EFA resulted in similar factor loadings of serum kynurenines across subcohorts. CE induced an acute increase in NeuroPro (0.421, 95% CI [0.216, 0.626], p < 0.001) in FunTraiMS. Twenty-four-week endurance exercise induced a chronic decrease in NeuroTox (-0.432, 95% CI [-0.786, -0.080], p = 0.019) in EXBRAIN. NeuroTox and NeuroPro are temporally stable constructs. NeuroPro shows acute responsiveness to strength-focused exercise. NeuroTox may be chronically diminished by endurance exercise. Determinants and the clinical relevance of changes in NeuroTox and NeuroPro warrant further investigation.
Date Issued
2026-07-29
Publication Type
Article
Subject(s)
600 Technology > 610 Medicine & health
Subjects
Exercise
•
Kynurenine
•
MS
•
Multiple sclerosis
•
Tryptophan
Language(s)
en
Author(s)
Kupjetz, Marie
Langeskov-Christensen, Martin
Riemenschneider, Morten
Inerle, Stefan
Ligges, Uwe
Patt, Nadine  
Graduate School for Health Sciences (GHS)  
Bansi, Jens
Gonzenbach, Roman Rudolf
McCann, Adrian
Ueland, Per Magne
Wences Chirino, Tiffany Yazmin
Joisten, Niklas
Hvid, Lars Grøndahl
Dalgas, Ulrik
Zimmer, Philipp
Additional Credits
Graduate School for Health Sciences (GHS)  
Journal
Brain, behavior, and immunity
ISSN
1090-2139
Access(Rights)
embargo
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