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  3. Genetic variants in SLC22A17 and SLC22A7 are associated with anthracycline-induced cardiotoxicity in children

Genetic variants in SLC22A17 and SLC22A7 are associated with anthracycline-induced cardiotoxicity in children

Details
Publisher DOI
10.2217/pgs.15.61
PubMed ID
26230641
Abstract
AIM

To identify novel variants associated with anthracycline-induced cardiotoxicity and to assess these in a genotype-guided risk prediction model.

PATIENTS & METHODS

Two cohorts treated for childhood cancer (n = 344 and 218, respectively) were genotyped for 4578 SNPs in drug ADME and toxicity genes.

RESULTS

Significant associations were identified in SLC22A17 (rs4982753; p = 0.0078) and SLC22A7 (rs4149178; p = 0.0034), with replication in the second cohort (p = 0.0071 and 0.047, respectively). Additional evidence was found for SULT2B1 and several genes related to oxidative stress. Adding the SLC22 variants to the prediction model improved its discriminative ability (AUC 0.78 vs 0.75 [p = 0.029]).

CONCLUSION

Two novel variants in SLC22A17 and SLC22A7 were significantly associated with anthracycline-induced cardiotoxicity and improved a genotype-guided risk prediction model, which could improve patient risk stratification.
Date Issued
2015-07
Publication Type
Article
Subject(s)
600 Technology > 610 Medicine & health
Subjects
anthracyclines
•
association study
•
cardiotoxicity
•
childhood cancer
•
pharmacogenomics
Language(s)
en
Author(s)
Visscher, Henk
Rassekh, S Rod
Sandor, George S
Caron, Huib N
van Dalen, Elvira C
Kremer, Leontien C
van der Pal, Helena J
Rogers, Paul C
Rieder, Michael J
Carleton, Bruce C
Hayden, Michael R
Ross, Colin J
Amstutz, Ursula  
Universitätsinstitut für Klinische Chemie (UKC)  
Canadian Pharmacogenomics Network for Drug Safety consortium, CPNDS
Additional Credits
Universitätsinstitut für Klinische Chemie (UKC)  
Journal
Pharmacogenomics
Publisher
Future Medicine
ISSN
1462-2416
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