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  3. Exaggerated pulmonary hypertension and right ventricular dysfunction in high-altitude dwellers with patent foramen ovale.
 

Exaggerated pulmonary hypertension and right ventricular dysfunction in high-altitude dwellers with patent foramen ovale.

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BORIS DOI
10.7892/boris.76410
Date of Publication
April 2015
Publication Type
Article
Division/Institute

Universitätsklinik fü...

Author
Brenner, Roman
Pratali, Lorenza
Rimoldi, Stefano
Universitätsklinik für Kardiologie
Murillo Jauregui, Carla Ximena
Soria Maldonado, Rodrigo
Universitätsklinik für Kardiologie
Rexhaj, Emrush
Universitätsklinik für Kardiologie
Salinas Salmón, Carlos
Villena, Mercedes
Romero, Catherine
Sartori, Claudio
Allemann, Yves
Universitätsklinik für Kardiologie
Scherrer, Urs
Universitätsklinik für Kardiologie
Subject(s)

600 - Technology::610...

500 - Science::570 - ...

Series
Chest
ISSN or ISBN (if monograph)
0012-3692
Publisher
American College of Chest Physicians
Language
English
Publisher DOI
10.1378/chest.14-1353
PubMed ID
25375664
Description
BACKGROUND

There is considerable interindividual variability in pulmonary artery pressure among high-altitude (HA) dwellers, but the underlying mechanism is not known. At low altitude, a patent foramen ovale (PFO) is present in about 25% of the general population. Its prevalence is increased in clinical conditions associated with pulmonary hypertension and arterial hypoxemia, and it is thought to aggravate these problems.

METHODS

We searched for a PFO (transesophageal echocardiography) in healthy HA dwellers (n = 22) and patients with chronic mountain sickness (n = 35) at 3,600 m above sea level and studied its effects (transthoracic echocardiography) on right ventricular (RV) function, pulmonary artery pressure, and vascular resistance at rest and during mild exercise (50 W), an intervention designed to further increase pulmonary artery pressure.

RESULTS

The prevalence of PFO (32%) was similar to that reported in low-altitude populations and was not different in participants with and without chronic mountain sickness. Its presence was associated with RV enlargement at rest and an exaggerated increase in right-ventricular-to-right-atrial pressure gradient (25 ± 7 mm Hg vs 15 ± 9 mm Hg, P < .001) and a blunted increase in fractional area change of the right ventricle (3% [-1%, 5%] vs 7% [3%, 16%], P = .008) during mild exercise.

CONCLUSIONS

These findings show, we believe for the first time, that although the prevalence of PFO is not increased in HA dwellers, its presence appears to facilitate pulmonary vasoconstriction and RV dysfunction during a mild physical effort frequently associated with daily activity.

TRIAL REGISTRY

ClinicalTrials.gov; No.: NCT01182792; URL: www.clinicaltrials.gov.
Handle
https://boris-portal.unibe.ch/handle/20.500.12422/138445
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