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  3. Immune disturbances in individuals with post-COVID syndrome are not characterized by enhanced SARS-CoV-2-specific immunity.

Immune disturbances in individuals with post-COVID syndrome are not characterized by enhanced SARS-CoV-2-specific immunity.

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DOI
10.48620/88161
Publisher DOI
10.1093/infdis/jiaf211
PubMed ID
40279368
Abstract
Background
Post-COVID syndrome (PCS) is characterized by persistent symptoms lasting beyond 12 weeks after SARS-CoV-2 infection. The underlying pathomechanims remain poorly understood.
Methods
We conducted detailed immunological analyses in 47 individuals with PCS, assessed >12 weeks post-acute SARS-CoV-2 infection, and compared them to 25 convalescent controls without symptoms. We performed immune phenotyping of T and B cell subsets, assessed SARS-CoV-2-specific responses using activation-induced marker (AIM) flow cytometry for T cells, and tetramer staining of spike-specific B cells. Cytokine levels in peptide-stimulated cell supernatants and plasma were quantified using a Luminex platform.
Results
PCS individuals exhibited reduced frequencies of AIM+ SARS-CoV-2-specific T cells (OX40+, PDL1+) and CD8 T cells (CD137+, CD69+) following petide stimulation with S- or N-Antigen, accompanied by diminished IFNγ and IL2 as measured in the cell culture supernatants. In contrast, non-virus specific T cell populations, including their memory differentiation, activation and helper cell differentiation status did not differ between the groups. PCS individuals showed a significant increase in total (CD19+) and activated B cells (CD86+, HLA-DR+), but not in SARS-CoV-2 spike-specific B cells (∼0.2% of total B cells). Plasma cytokine analysis revealed elevated markers associated with vascular damage and inflammation in PCS individuals.
Conclusion
Persistent immune disturbances in individuals with PCS are characterized by reduced SARS-CoV-2-specific T cell responses, increased B cell activation, and altered inflammatory and vascular biomarkers. These findings provide insights into the underlying mechanisms of PCS and may contribute to biomarker discovery and therapeutic development.Trialnumber: DRKS00030974registry's URL: https://www.bfarm.de/EN/BfArM/Tasks/German-Clinical-Trials-Register/_node.html.
Date Issued
2025-07-30
Publication Type
Article
Subject(s)
600 Technology > 610 Medicine & health
Subjects
Sars-Cov2-specific immunity
•
inflammation
•
long COVID
•
post-COVID syndrome
Language(s)
en
Author(s)
Ober, Veronica
Völk, Felix
Sbierski-Kind, Julia
Gruener, Eva
Stirner, Renate
Reiling, Gabriele
Feldmann, Svenja
Ibarra, Gerardo
Seybold, Ulrich
Stubbe, Hans
Adorjan, Kristina  
University Hospital of Psychiatry and Psychotherapy  
University Hospital of Psychiatry and Psychotherapy  
Bogner, Johannes R
Roider, Julia
Additional Credits
University Hospital of Psychiatry and Psychotherapy  
Journal
The Journal of Infectious Diseases
Publisher
Oxford University Press
ISSN
1537-6613
0022-1899
Access(Rights)
open.access
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